If you’re a PhD degree holder and seeking postdoctoral fellowships, Aalborg University, Denmark has several online applications open. Explore the opportunities across diverse research areas and submit your application soon.
1. Postdoctoral Job
Summary of Postdoctoral Fellowship:
Research areas will be within comprehensive CFD modelling of oxy-fuel combustion of biomass(or biomass/ coal co-firing) in a 30MW circulating fluidized bed(CFB) boiler, CFD validation with existing experimental data in the boiler, and then CFD-based virtual testing of this boiler for advancing oxy-fuel CFB technology(e.g., new gas-staging, oxygen-staging, oxy-steam combustion). It will be exciting to closely collaborate with highly competent partners across the EU on different oxy-combustion CCS technologies such as oxy-CFB, CLC, and oxy-MILD.
Application Deadline: 28.07.2024
2. Postdoctoral Job
Summary of Postdoctoral Fellowship:
You will join the ambitious European collaboration ARISTOTELES(Applying artificial intelligence to define clinical trajectories for personalized prediction and early detection of comorbidity and multimorbidity patterns). Further, you will contribute to ARISTOTELES’ aim to leverage artificial intelligence for the personalized prediction and early detection of comorbidity and multimorbidity patterns in atrial fibrillation patients. The key responsibility of the position is to structure atrial fibrillation patient’s health data for machine learning algorithms(feature engineering), contribute to the development of machine learning based algorithms for predicting complications to atrial fibrillation, and implementation of predictive models in the clinic.
Application Deadline: 28.06.2024
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3. Postdoctoral Job
Summary of Postdoctoral Fellowship:
A Postdoc position is available to explore the enigmas of intracellular signal transduction with a focus on the calcium(Ca2+) feedback loop in IP3/Ca2+-signaling. IP3 stimulates the release of Ca2+ stored in the endoplasmic reticulum through IP3 receptors(IP3Rs). A critical aspect of Ca2+-signaling is the ability of Ca2+ to self-regulate by feedback mainly mediated by the Ca2+-binding protein calmodulin(CaM). When Ca2+ binds, the shape of CaM changes, enabling it to modulate the activity of the IP3Rs. Mutations in CaM cause deadly cardiac arrhythmias and neurological disorders, much like diseases caused by perturbations of the InsP3/Ca2+ signaling pathways. Despite the universality and intersection of these pathways, the mechanisms by which CaM regulates the IP3Rs remain unresolved.
Application Deadline: 27.06.2024
